- Grammy winning singer-songwriter, Stevie Nicks snorted so much cocaine and became so addicted to the drug that she had to be shadowed to keep from falling off stage when performing and needed to have someone tuck her into bed at night
- The Queen of Rock and Roll in the 1970s and 1980s not only had a huge hole in her nose from the cocaine, but she was warned of the imminent possibility of a brain hemorrhage if she kept up her high level of consumption
- But it was the shocking rumors that she had reverted to using the devil’s dandruff in her vagina and rectum for the ultimate high that was the eventual motivation for her to go into rehab in 1986 at the Betty Ford addiction treatment center in Minnesota
- The Fleetwood Mac singer admitted: “You could put a big gold ring through my septum. It affected my eyes, my sinuses. It was a lot of fun for a long time because we didn’t know it was bad. But eventually it gets hold of you, and all you can think about is where your next line is coming from”
- “All of us were drug addicts. But there was a point where I was the worst drug addict. I was a girl, I was fragile, and I was doing a lot of coke and I was in danger of brain damage,” she told author Stephen Davis for his upcoming book, Gold Dust Woman: The Biography of Stevie Nicks
- Ronald and Carla Hiers were seen writhing around in a Memphis street after injecting heroin
- The pair had been together for 20 years when they were filmed last October
- After being saved by paramedics, Carla was arrested on outstanding charges
- Ronald went home and tried to kill himself with 48 Xanax pills and more heroin
- His estranged daughter then persuaded him to enter a rehab center
- They are no longer together and live in separate states
- Both have completed separate treatment plans and are now clean and sober
A husband who was filmed overdosing with his wife after shooting up heroin in one of the most symbolic videos of America’s drugs epidemic last year has shared new details of their crippling addiction and how they overcame it.
On October 3, 2016, Ronald and Carla Hiers were filmed crawling along the sidewalk and passed out at a bus stop in Memphis after injecting heroin in the bathroom of a Walgreen’s nearby.
Footage of them writhing around in broad daylight as they reeled from the drug’s effects attracted millions of views on Facebook.
The couple have since completed separate rehab programs and are no longer living together or even in the same state.
His addiction began at 13 when he and his friends started sniffing paint. They graduated on to marijuana and then began using harder drugs when he was around 18.
Not long after the video went viral last year, Ronald was admitted to Turning Points in Tennessee.
Carla was released from jail several weeks later and went to a separate facility in Massachusetts.
Mom's Personality Changed - Xanax To Blame? « on: July 19, 2017, 01:21:44 am »
About 2 years ago my mom’s whole personality changed. Her OCD symptoms got much worse, she says incredibly rude things she NEVER would’ve said before & her memory has gone down the toilet. Her routines & rituals are set in stone & she won’t deviate from them. She leaves the stove on 2x per month on average now. She started taking Xanax (1mg-2mg per week on average) around the same time her personality changed. The worst memory lapses–such as leaving the stove on & forgetting basic words–always happen the day after she takes her Xanax dose. She’s also excessively tired the day after taking Xanax. Worst of all, she denies any change in her personality & gets defensive even talking about it.
Could using Xanax one night per week be sufficient to affect someone’s mood, cognition & anxiety levels to this degree? She’s had a CT scan of her brain at my insistence to see if there was any sign of previous strokes or other issues, and it came back relatively normal. (Some age-related shrinkage & atherosclerosis). The doctor did not seem concerned about it, though it was an ear/nose/throat doctor rather than a neurologist. She’s 65 w/ no family history of Alzheimer’s but at moderate risk for stroke. She’s lost a good bit of vision & hearing due to age, so it’s hard to tell whether she’s doing things like leaving the stove on because of those impairments or something more sinister.
I’m not asking anyone to definitively diagnose her here; just wanted to see if anyone’s experienced this degree of side effects from low dose, once weekly benzo use. (Personally, if I took a benzo or barbiturate one time per week, that would be sufficient to cause rebound mood/anxiety problems but I don’t know how common that is). My mom absolutely refuses to go to doctors, so that’s out of the question. She swears up and down she doesn’t take Xanax more frequently than 1x per week, which I believe because she’s so regimented & afraid of drug addiction, but she could be taking it more often. I’m at a loss.
OCD runs in our family, as do other mental illnesses. I’m absolutely terrified it’s dementia, which would probably cause me to kill myself or be institutionalized because I could not handle that. Cancer is preferable to dementia. I’m praying it’s the Xanax at this point because the alternatives are so awful.
Re: Mom's Personality Changed - Xanax To Blame? « Reply #1 on: July 19, 2017, 01:31:29 am »
I am curious to why she takes one Xanax a week. It seems like she would be in perpetual withdrawal. Her symptoms could well be withdrawal symptoms. It certainly causes brain fog and forgetting things.
Re: Mom's Personality Changed - Xanax To Blame? « Reply #2 on: July 19, 2017, 02:26:13 am »
Also, you might want to take into consideration paradoxical reactions:
Benzodiazepine Side Effects: http://www.benzo.org.uk/sidefx.htm
So-called “Paradoxical” Effects
According to Professor Malcolm Lader, 5% of those using benzodiazepines may be affected by so-called “paradoxical” reactions in response to the drugs rather than the desired tranquillising effects. Such reactions include increased aggressiveness (in some individuals even violent behaviour), depression (with or without suicidal thoughts or intentions), and sometimes personality changes.
Paradoxical” side effects occur in all age groups but are more likely to be found in children and in the elderly where they may be fairly frequent yet erroneously diagnosed as various psychiatric disorders. The risk of such reactions is generally greater with short-acting compounds but may occur with all benzodiazepine drugs. It is important to remember that the “paradoxical” reactions can also be encountered in short-term use and, in rare cases, even following the first ingestion of the drug.
Cognitive Side Effects
Memory functioning is markedly and measurably impaired, especially the ability to store acquired knowledge into long-term memory. This memory impairment is highly relevant to students. The risk of acute amnesia is more pronounced with short-acting drugs. Ativan (lorazepam), Halcion (triazolam), Xanax (alprazolam) and Rohypnol (flunitrazepam) are especially likely to induce such memory impairment.
Re: Mom's Personality Changed - Xanax To Blame? « Reply #3 on: July 19, 2017, 03:00:09 am »
Here’s some more info on this: http://w-bad.org/paradoxical/
I hope it’s the Xanax and your Mom might agree to stop taking it to test out whether this is the case.
Re: Mom's Personality Changed - Xanax To Blame? « Reply #4 on: July 20, 2017, 06:41:33 am »
Thank you so much
She’s very regimented due to OCD (which has gotten worse with age/Xanax use). She goes shopping one day per week, so the Xanax is to help her sleep the night before. I’ve seen her take it twice in a week when she had other things to do, so she may be taking it more often than 1x per week.
She has an endless supply obtained from…well, let’s just say this isn’t coming from a doctor. She’s had prescriptions for it in the past & even forged one prescription to have 4 refills instead of zero. So that tells me she’s got some kind of issue right there. But she could’ve just forged it because she hates going to the doctor. Either way, it’s a crime & she could’ve gone to jail for it. But back then she didn’t take Xanax as often as she does now.
I’m very familiar with rebound symptoms like anxiety & low mood, as I get them after a single use of barbiturates, benzos or Ambien. Thanks again for the links & quotes. That gives me some peace of mind. The part about short-acting benzos being even more likely to cause issues is especially comforting. I’ve tried explaining rebound effects to her before, but she doesn’t believe that can really happen even after SEEING what a single dose of benzos does to me the following day. She once became manic from a single dose of Valium too.
Ugh. This is all very frustrating since I now live with her. But if it’s “just” the benzos causing her insanity, that would be a good thing
Contractor stole my Xanax yesterday... « on: July 08, 2017, 02:20:00 pm »
Which puts me in a state of panic. I just had my prescription refilled 3 days ago, 2 days ago I put the prescription in its normal place in my medicine cabinet and put a small amount I keep with me in another bottle.
Had a local contractor come to my house to give me a quote on some work. He had to use the bathroom twice within 15 minutes – he used both bathrooms instead of the just guest bathroom in the front of the house used the first time – he had no business even going into my bedroom to get to my master-bath. I was in the crawl space when he yelled out to me that he was going to use the bathroom again – and he helped himself to our master bathroom.
Long story short, he stole my Xanax out of my medicine cabinet. I have filed a police report, unfortunately I don’t keep a camera zeroed in on the bathroom door so it’s my word against his. But knowing I took the medication that morning and it was no longer there as soon as he left – there is NO DOUBT.
I’m prescribed 4/day but am down to 3. I am tapering with my doctors knowledge on my own schedule – of course I am afraid he and all other doctors have heard this excuse a million times simply to get more meds. At least I have a police report and my wife as a character witness but still don’t know if that will help. One time when my doctor allowed for an early refill but the pharmacist actually overturned his decision.
I can’t believe the nerve of some people.
« Last Edit: July 08, 2017, 02:25:56 pm by [Buddie] »
Re: Benzodiazepine information coalition: does this place exist? « Reply #42 on: June 23, 2017, 01:34:52 pm »
wanted to ask you your thoughts about the difference between the benzo’s “withdrawal” damage/injury iatrogenic illness, benzo discontinuation syndrome or whatever you want to call it and opiate addiction? because i know for me that opiates caused a craving where as i never craved benzos. you know what i mean/ there’s just a difference in these two drugs. i ponder this a lot.
My only experience with opiates has been the few times I have taken hydorcodone or percocet. I remember having this “wow” feeling the first time I took them so I can see how they could rope someone in.
It seems like benzo withdrawal (or whatever you want to call it) is a completely different animal. They don’t create physical cravings for most people but the severity and duration of the damage they cause to the body seems to be worse in general. I wouldn’t want to find myself addicted to opiates but if I could trade that for what I have experienced over the past 6.5 years from benzos I would do it in a heartbeat. At least if the bulk of your problem is staying off the drugs you might have a fighting chance.
Denying that benzos create physical craving in most people is simply addict shaming, […]. It’s intolerance and approaches bigotry. It shuts down conversation about benzos and is seen by many professionals and lay people as denial. Denial is a hallmark of addiction. This conversation is unpopular here and it’s not my fight. My fight is overcoming a lifetime of taking these pills and regaining my life.
Benzo craving is prevalent here at bb’s and can be seen in the vast majority of early posts before people are indoctrinated into the bb’s culture. Even then the veterans display the cravings in many posts but rationalize it away as specific symptoms. Rationalization is another hallmark of addiction.
Addiction doesn’t discriminate. Humans do!
The term addiction fits for most of us. Breaking the symptoms of withdrawal down into minute details is again denial and rationalization best used only in support groups. The broad picture of the minute details supports an addiction definition and paradigm. Post withdrawal syndrome and the time it takes for the small subsection of us to recover is a whole other discussion.
Refusal by some members here to accept that many many people here are addicted despite the overwhelming evidence otherwise shuts down healthy and critical analysis of our issues. I understand why people don’t want to be associated with addiction. But the very nature of being here at bb’s involved in support for getting off benzos suggests we are associated with addiction. That’s how most of the real world understands this.
I agree that the definition af addiction carries with it many awful preconceptions that it shouldn’t, but that definition is embedded into the worldwide human culture. Overcoming those biases held by everyone who is culturally assimilated is a tall task. Overcoming those biases in our worlds cultures changes the conversation for those of us who are trying to recover to something else.
I apologize to anyone that is offended by this post. It’s not my intent to offend but out there in the real world most people I run into only know this as addiction. It’s how they understand the issue.
Addict shaming sucks where ever you find it.
« Last Edit: June 23, 2017, 02:46:26 pm by [Buddie] »
The anti-psychiatry cult venerates Nicks as an anti-benzo apostle yet Stevie was an out of control drug addict:
- Fleetwood Mac singer Stevie Nicks was so addicted to cocaine, alcohol and Quaaludes she blacked out and nearly overdosed repeatedly
- She wore gold and turquoise bottle inlaid with diamonds around her neck so she was never without coke
- To avoid body searches by customs in Europe, they hired Hitler’s private rail car complete with the elderly attendant who served the Fuhrer
She quickly descended into drug hell and became addicted to cocaine, alcohol, Quaaludes to sleep, and cigarettes – until her system broke down and she started having nosebleeds, falls on stage, blackouts and near overdoses.
She bought $1 million worth of cocaine and it burned a hole in her nose the size of a dime. Rumors spread that she had to have the drug blown up her derriere by an assistant.
“There was no way to get off the white horse and I didn’t want to,” the now 66-year-old Nicks said.
She only slowed down her drug consumption when her doctor warned her she was risking permanent mental and physical damage as well as heading for a brain hemorrhage or an early grave.
The group called for an intervention and saved her life by urging her to check in to the Betty Ford Center.
Can anyone blame Big Pharma, or psychiatry, for that?
In a March 2017 Rolling Stone interview, instead of advising her younger self never to take illegal drugs, and thereby help other addicts, Nicks irresponsibly blames the psychiatrist who tried to help her recover:
What advice would you give to your younger self?
“How about my early-forties self? That’s when I walked out of Betty Ford after beating coke. I spent two months doing so well. But all my business managers and everyone were urging me to go to this guy who was supposedly the darling of the psychiatrists. That was the guy who put me on Klonopin. This is the man who made me go from 123 pounds to almost 170 pounds at five feet two. He stole eight years of my life.”
Look at what this poor, brainwashed, slob at Benzo Buddies says about Nicks:
Resist « on: May 24, 2017, 02:51:03 am »
Start fighting back folks. No more suicides. Lost jobs and homes. No more drugged toddlers and babies, elderly and infirm. Come on guys! This is a grass roots effort! If Stevie Nicks still has the balls to stand up against big pharma and the drug dealers that push their poisons, so do you!
Love you all,
Talk about delusional.
“We were all selling drugs by the time we were 12, or doing them… pot or pills or anything that was easily available.” – Chris Cornell, 1994 Rolling Stone interview
How can we get this message of benzo dangers out? Soundgarden - Cornell- Ativan « on: May 19, 2017, 01:42:27 pm »
Lawyer Kirk Pasich said Cornell, a recovering addict, had a prescription for Ativan, which he said has a range of side effects, including suicidal thoughts.
Major side effects associated with Ativan use include confusion, depression, and memory loss, according to AmericanAddictionCenters.org.
I would suspect he was addicted to ativan and didn’t know how he was going to beat the dependency. Wife said he took a few extra before concert.
USA does not take Benzo addiction seriously. Only opiod.
- Ask a spouse or parent who has struggled for years to help a drug user and you might hear that an addict is someone who betrays you and takes whatever they can get, who bankrupts you and breaks your heart.
- Ask a law enforcement officer who tried to help at first but then gave up because of the overwhelming extent of the problem and he might talk about the hopelessness of even making an effort.
- Ask a doctor who has seen too many patients scream at him and his staff if he fails to give them the pills they want and he may rant about how horrible and dangerous “these people” are.
- Ask an emergency room nurse and she might wave her hand in despair of ever being able to do more than keep a person alive so he can use drugs again the next night.
- Ask someone who tried to help an addicted person again and again but then gave up in disgust when the person always returned to the bottle or the needle, despite that offer of help. Perhaps he can’t be blamed for concluding that an addict is someone who can’t be helped, who is hell-bent on destroying himself, who is degraded all the way down to his soul.
Recreational use of ketamine
Ketamine is a prescription anesthetic that is federally regulated (e.g., U.S. Schedule III, U.K. Class B) that functions as a dissociative anesthetic, and so has seen use as a recreational drug. Originating in the United States in the 1970s, the recreational use of ketamine has since spread to Europe, Canada, Asia, and Australia. Attempts are made to use the drug at sub-anesthetic doses; contexts for use include both private settings and at club venues (raves and parties), where it initially gained popularity. Despite its emergence as a club drug, users may eventually relegate their use to more private settings
Ketamine interacts with a variety of other drugs, most pronounced with alcohol, opioids (potentiation), and barbiturates, and with drugs that increase blood pressure (e.g., stimulants, SNRI antidepressants, and especially MAOIs). The latter may have an additive effect on the user’s blood pressure, causing tachycardia, palpitations and potentially serious arrhythmias.[not verified in body] Ketamine use as a recreational drug has been implicated in a small but greatly exaggerated number of annual deaths, the majority of which are youth or young adults, which have, taken together, led to increasing stringency of its regulation worldwide.
As a consequence of its drug interactions and adverse effects, including the ability to cause confusion and amnesia, and adverse reactions that occur during emergence from anaesthesia, some cases are known from the media that involved irresponsibly high dosages and accidents in people who were not prepared for the experience and/or took other drugs at the same time, despite ketamine being a physically very safe substance in comparison to other psychoactives like opioids or even alcohol. But ketamine can leave users vulnerable to date rape (i.e., because of the associated confusion and amnesia).
Due to the complexity of its chemical synthesis, ketamine supplies for recreation use must be diverted from licit medical sources, though there have been reports of industrial-scale illicit ketamine manufacture in China and India.
Ketamine’s use as a recreational drug has contributed to more than 90 fatalities—the majority among young adults—in England and Wales in the years of 2005-2013, including accidental poisonings, drownings, and traffic accidents. This has led to its increasingly stringent regulation (e.g., upgrading ketamine from a Class C to a Class B banned substance in the U.K.).
Ketamine may increase the effects of other sedatives, including, but not limited to: alcohols, benzodiazepines, opioids, and barbiturates. Other drugs which increase blood pressure may interact with ketamine, having an additive effect on blood pressure; such agents include stimulants, SNRI antidepressants, and MAOIs. Increase blood pressure and heart rate, palpitations, and arrhythmias may be potential effects.
Ketamine is generally safe even for those critically ill, when administered by trained medical professionals. The dosages used recreationally are somewhat lower than a fully anaesthetic dosage, making the substance physically relatively safe in comparison to other psychoactive drugs. Still, even in these cases, there are known side effects that include one or more of the following:
- Cardiovascular: abnormal heart rhythms, slow heart rate or fast heart rate, high blood pressure or low blood pressure; and
- Central nervous system: increased intracranial pressure (ICP), leading to intracranial hypertension.
In addition there are dermatologic adverse reactions (ARs; transient erythema, transient morbilliform rash), gastrointestinal ARs (anorexia, nausea, increased salivation, vomiting), neuromuscular and skeletal ARs (Increased skeletal muscle tone, i.e., tonic-clonic movements), ocular ARs (double vision, increased intraocular pressure, nystagmus), respiratory ARs (airway obstruction, apnea, increased bronchial secretions, respiratory depression, laryngospasm), as well as local pain or exanthema (e.g., at injection sites) and possible anaphylaxis and dependence.
In 10-20% of patients at anesthetic doses, adverse reactions are experienced that occur during emergence from anesthesia, reactions that can manifest as seriously as hallucinations and delirium. These reactions may be less common in some patients subpopulations, and when administered intramuscularly, and can occur up to 24 hours postoperatively; the chance of this occurring can be reduced by minimizing stimulation to the patient during recovery and pretreating with a benzodiazepine, alongside a lower dose of ketamine. Patients who experience severe reactions may require treatment with a small dose of a short- or ultrashort-acting barbiturate.
Ketamine produces effects similar to phencyclidine (PCP) and dextromethorphan (DXM). Unlike these other well-known dissociatives, ketamine is very short-acting, its hallucinatory effects lasting tens of minutes when inhaled (insufflated) or injected, and hours when ingested. With ketamine, intensities of hallucinations are dose-dependent. Like other dissociative anaesthetics, hallucinations caused by ketamine are fundamentally different from those caused by serotonergic psychedelic (classic) hallucinogens.
The specific dissociative state produced by ketamine is characterised by a sense of detachment from one’s physical body and the external world that is known as depersonalization and derealization. At sufficiently high doses, users may experience what is called the “K-hole”, a state of extreme dissociation with phenomenology of schizophrenia (e.g., visual and auditory hallucinations).
John C. Lilly, Marcia Moore and D. M. Turner (amongst others) have written extensively about their own entheogenic use of and psychonautic experiences with ketamine. Both Moore and Turner died prematurely (due to hypothermia and drowning respectively) during presumed unsupervised ketamine use.
As the recreational dosages used are always below the fully anaesthetic threshold and thus lower than those medically administered, the term ‘overdose’ needs to be seen a bit differentiated from other drugs. While with continued use, people may build up some degree of tolerance, leading to the use of higher dosages than medically advised, it is technically somewhat difficult to overdose on ketamine. An even higher dosage will just lead to full anaesthesia, amnesia, but no physical danger from the drug itself as long as the environment isn’t dangerous. The user will become catatonic when fully dissociated, not experiencing any pain but also unable to move his/her body.
There is no known effective antidote used to treat ketamine overdose, and treatment generally focuses on the maintenance of respiratory and circulatory function until the patient is capable of breathing under their own power and all cardiac abnormalities have subsided. Unlike many other anaesthetics, ketamine has a minimal effect on respiratory drive and tidal volume; while pulse oximetry is always essential it rarely drops enough to require mechanical ventilation or supplemental oxygen, except in the most massive of overdoses and in cases of mixed-drug-overdose.
Verbal reassurance and a calming environment (i.e. dim lights, calming music, and the presence of supportive individuals in the room) should be provided when possible to calm the patient and/or prophylacticly to prevent agitation. Occasionally it may become necessary to restrain highly agitated patients during recovery should they become violent, experience panic attacks, or otherwise present a threat to themselves or others.
Even after all vital signs have normalized and the patient appears functional it is advised to require the patient to be driven home as residual impairment of motor skills may persist for up to a day after apparent resolution.
Ketamine’s potential for dependence has been established in various operant conditioning paradigms, including conditioned place preference and self-administration; further, rats demonstrate locomotor sensitization following repeated exposure to ketamine. Increased subjective feelings of ‘high’ have been observed in healthy human volunteers exposed to ketamine. Additionally, the rapid onset of effects following smoking, insufflation, and/or intramuscular injection is thought to increase the drug’s recreational use potential. The short duration of effects promotes bingeing, tolerance can develop, and withdrawal symptoms, including anxiety, shaking, and palpitations, may be present in some daily users following cessation of use.
Due to its primary NMDA-antagonist effect, sudden withdrawal in severely addicted users will result in overexcitability, manifesting as increased sensitivity to stress, anxiety and pain. There are speculations about possible excitotoxicity resulting from the rebound surge in glutamate, but this has not yet been proven or disproven in humans and it doesn’t seem to be a huge concern in healthy adults. Unlike GABAergic sedatives however, overexcitation secondary to ketamine withdrawal is not life-threatening as long as no underlying seizure disorders are present and even very tolerant users will likely suffer, at worst, only minor neurological sequela following the abrupt discontinuation of the drug. Some titration or the administration of anti-excitatory agents like memantine could be of benefit.
Ketamine can cause a variety of urinary tract problems that are more likely to occur with heavier and/or higher dosed use, especially in those not watching for a healthy lifestyle, according to a UK study.
See also: K-Hole (“K-hole” is a slang term for the subjective state of dissociation from the body commonly experienced after sufficiently high doses of the dissociative anesthetic ketamine. This state may mimic the experiences such as catatonic schizophrenia, out-of-body experiences (OBEs) or near-death experiences (NDEs), and is often accompanied by feelings of extreme derealization, depersonalization and disorientation, as well as temporary memory loss and vivid hallucinations.)